Gastric Diospyrobezoar Dissolution with Ingestion of Diet Soda and Cellulase Enzyme Supplement

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Gastric Diospyrobezoar Dissolution with Ingestion of Diet Soda and Cellulase Enzyme Supplement

Diospyrobezoars are a subtype of phytobezoars caused by excessive consumption of persimmons, which contain large amounts of tannins. In contrast to phytobezoars, diospyrobezoars have a harder consistency than other bezoars, making them more difficult to break up both chemically and endoscopically. We have previously reported successful dissolution of phytobezoars with diet soda and cellulase. A...

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Gastric phytobezoar dissolution with ingestion of diet coke and cellulase.

Address correspondence to: Dr. Mark B. Pochapin, NYU Langone Medical Center, 522 First Avenue, Suite 901B, New York, NY 10016; Tel: 646-501-6760; Fax: 646-501-6767; E-mail: [email protected] A bezoar is an indigestible mass of material—such as hair, food, seeds, or another ingested substance—found in the gastrointestinal tract.1 A phytobezoar, the most common type of bezoar, is composed o...

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Diet soda and insulin spikes

I have heard that the main reason why diet drinks, like a diet soda, can be bad on a diet, is that it can spike insulin levels and then your body expects sugar that it does not get. I have taken to the habit of only occasionally having diet drinks with meals, so that any increase in insulin is actually met with food in my system. My question is whether or not this is a good/workable strategy, o...

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Ingestion of Diet Soda Before a Glucose Load Augments Glucagon-Like Peptide-1 Secretion

OBJECTIVE The goal of this study was to determine the effect of artificial sweeteners on glucose, insulin, and glucagon-like peptide (GLP)-1 in humans. RESEARCH DESIGN AND METHODS For this study, 22 healthy volunteers (mean age 18.5 +/- 4.2 years) underwent two 75-g oral glucose tolerance tests with frequent measurements of glucose, insulin, and GLP-1 for 180 min. Subjects drank 240 ml of die...

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ژورنال

عنوان ژورنال: ACG Case Reports Journal

سال: 2017

ISSN: 2326-3253

DOI: 10.14309/crj.2017.90